Obesity is a chronic inflammatory disease which is associated with activation of HIF signaling in various tissues. Furthermore, the HIF-driven pathophysiological mechanisms that are involved in the progression of fatty liver to non-alcoholic steatohepatitis (NASH) and hepatocellular carcinoma (HCC) remains to be elucidated. The upstream pathways and signaling cues that are involved in the activation of HIF, independent of intracellular oxygen levels, remain unknown. However, chronic activation of HIF is shown to be detrimental and can lead to the numerous pathologies including fatty liver. A highly conserved transcription factor, hypoxia-inducible transcription factor (HIF) orchestrates the adaptive response to low oxygen levels (hypoxia). Adaptation to the changes in intracellular oxygen levels is a challenge to the tissues particularly liver, a hub of all metabolic responses.
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The common mode input range includes the negative supply, thereby eliminating the necessity for external biasing components in many applications. The amplifier can operate at supply voltages as low as 3.0 V or as high as 32 V. It has several distinct advantages over standard operational amplifier types in single supply applications. The LM358 opamp features low power drain, a common mode input voltage range extending to ground/VEE, and single supply or split supply operation. Operation from split power supplies is also possible and the low power supply current drain is independent of the magnitude of the power supply voltage. The LM358 is available in a chip sized package (8-Bump micro SMD) using National’s micro SMD package technology. 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